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STEMI vs NSTEMI
Acute coronary syndromes
FeatureSTEMINSTEMI
ECGST elevationST depression / T-wave inversion / normal
TroponinElevatedElevated
OcclusionCompletePartial
Urgent RxPrimary PCI (<120 min)Medical management first
AntiplateletAspirin + TicagrelorAspirin + Ticagrelor
AngiographyImmediateWithin 72h (or sooner if high risk)
ThrombolysisIf PCI not availableNOT indicated
💡 Key point: Both have raised troponin. The difference is the ECG. STEMI = ST elevation = emergency PCI. NSTEMI = no ST elevation = medical management first.
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Left vs Right Heart Failure
Different sides, different symptoms
FeatureLeft Heart FailureRight Heart Failure
CauseIHD, hypertension, aortic/mitral valveLeft HF (most common), COPD, PE, pulm HTN
CongestionPulmonary (lungs)Systemic (body)
SymptomsBreathlessness, orthopnoea, PND, coughAnkle oedema, ascites, hepatomegaly, raised JVP
SignsBibasal crackles, S3 gallop, tachycardiaPitting oedema, hepatomegaly, raised JVP
CXRPulmonary oedema, cardiomegaly, effusionsMay be normal
💡 Remember: Left = Lungs. Right = Rest of body. Most common cause of right HF is left HF.
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Asthma vs COPD
Obstructive airway diseases
FeatureAsthmaCOPD
AgeYoung (<40)Older (>40)
SmokingNot alwaysAlmost always
ReversibilityFully reversiblePartially reversible
FEV1/FVC<0.7 (reversible with bronchodilator)<0.7 (persistent)
Diurnal variationYes (worse morning/night)No
AtopyCommon (eczema, hayfever)Uncommon
First-lineSABA + low-dose ICSSABA/SAMA PRN → LABA/LAMA
O2 target94-98%88-92%
⚠️ COPD patients: Target SpO2 88-92%. High-flow O2 can suppress respiratory drive and worsen CO2 retention.
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Type 1 vs Type 2 Diabetes
Different causes, different management
FeatureType 1Type 2
CauseAutoimmune beta cell destructionInsulin resistance + relative deficiency
AgeYoung (<30 usually)Older (>40, increasingly younger)
OnsetAcute (days-weeks)Gradual (months-years)
WeightOften thin/normalOften overweight/obese
KetosisCommon (DKA)Rare (HHS instead)
C-peptideLow/absentNormal/high
AntibodiesGAD, IA-2, ZnT8None
TreatmentInsulin (always, lifelong)Metformin → add agents → insulin
💡 Key point: T1DM = absolute insulin deficiency (autoimmune). T2DM = insulin resistance. Both cause hyperglycaemia but management is very different.
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Ischaemic vs Haemorrhagic Stroke
Critical to differentiate before treatment
FeatureIschaemic (85%)Haemorrhagic (15%)
CauseThrombus or embolus blocking arteryVessel rupture (intracerebral or subarachnoid)
Risk factorsAF, carotid stenosis, hypertensionHypertension, anticoagulants, AVM, aneurysm
OnsetSudden focal deficitSudden headache + focal deficit
CT headMay be normal early (hypodense later)Hyperdense (bright white) immediately
TreatmentThrombolysis (alteplase <4.5h) or thrombectomyReverse anticoagulation, BP control, neurosurgery
Aspirin300mg after CT excludes bleedCONTRAINDICATED
⚠️ MUST do CT head before any treatment. Giving thrombolysis to a haemorrhagic stroke is fatal. CT first, always.
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Crohn's vs Ulcerative Colitis
Inflammatory bowel diseases
FeatureCrohn's DiseaseUlcerative Colitis
LocationMouth to anus (anywhere)Rectum → colon only (continuous)
PatternSkip lesions (patchy)Continuous inflammation
DepthTransmural (full thickness)Mucosa/submucosa only
DiarrhoeaOften non-bloodyBloody diarrhoea + mucus
PainRIF pain (terminal ileum)LIF pain, tenesmus
ComplicationsFistulae, strictures, abscessesToxic megacolon, colorectal cancer
HistologyGranulomas, transmural inflammationCrypt abscesses, pseudopolyps
SmokingMakes it WORSEProtective (but dont recommend!)
SurgeryNot curative (recurs)Curative (colectomy)
💡 Mnemonic: Crohn's = CHRISTMAS: Cobblestoning, High temp, RIF pain, Intestinal fistulae, Skip lesions, Transmural, Mouth-to-anus, Abscess, Strictures.
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ACE Inhibitors vs ARBs
When to use which
FeatureACE Inhibitor (-pril)ARB (-sartan)
MechanismBlocks ACE enzymeBlocks AT1 receptor
ExampleRamipril, LisinoprilLosartan, Candesartan
CoughYes (bradykinin accumulation)No (no effect on bradykinin)
AngioedemaRare but seriousVery rare
HyperkalaemiaYesYes
First-line?Yes (HF, diabetes, CKD)If ACEi not tolerated (cough)
PregnancyContraindicatedContraindicated
MonitoringU&Es before + 1-2 weeks afterSame
💡 Key point: ACEi first-line. If dry cough → switch to ARB. Never combine ACEi + ARB (ONTARGET trial = harm).
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Upper vs Lower GI Bleed
Location changes everything
FeatureUpper GI BleedLower GI Bleed
LocationAbove ligament of TreitzBelow ligament of Treitz
PresentationHaematemesis, melaena, coffee-ground vomitFresh red blood PR, maroon stool
Common causesPeptic ulcer, varices, Mallory-WeissDiverticulosis, colorectal cancer, haemorrhoids
InvestigationOGD within 24hColonoscopy, CT angiography
ScoringGlasgow-Blatchford (pre), Rockall (post)No standard score
UreaRaised (digested blood)Normal
💡 Key point: Melaena (black tarry stool) = upper GI bleed. Fresh red blood PR = usually lower. Raised urea with normal creatinine = upper GI bleed.
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Hypothyroid vs Hyperthyroid
Too little vs too much
FeatureHypothyroidHyperthyroid
CauseHashimoto's (autoimmune), post-surgery, iodine deficiencyGraves' disease, toxic nodule, thyroiditis
TSH↑ High↓ Low
T4/T3↓ Low↑ High
WeightWeight gainWeight loss
Heart rateBradycardiaTachycardia, AF
TemperatureCold intoleranceHeat intolerance
SkinDry, coarse, puffyWarm, sweaty, thin
MoodDepression, fatigueAnxiety, irritability, tremor
BowelsConstipationDiarrhoea
TreatmentLevothyroxine (lifelong)Carbimazole → radioiodine or surgery
💡 Remember: Hypo = everything SLOW. Hyper = everything FAST. TSH is the first-line screening test.
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Sensitivity vs Specificity
Understanding diagnostic tests
FeatureSensitivitySpecificity
QuestionHow good at detecting disease?How good at ruling out disease?
FormulaTP / (TP + FN)TN / (TN + FP)
High value meansFew false negativesFew false positives
Best forScreening testsConfirmatory tests
If negativeRules OUT disease (SnNOut)
If positiveRules IN disease (SpPIn)
💡 SnNOut: High Snensitivity + Negative result = rules Out. SpPIn: High Specificity + Positive result = rules In.